Session Details

TAFI

Monday, 22 March 2004 14:00 -15:30

Co-Chair Laszlo Bajzar Henderson Research Centre / McMaster UniversityCanada

Inhibition of Activated Thrombin-activatable Fibrinolysis Inhibitor (TAFIa) Improves Efficacy of Tissue Plasminogen Activator (tPA) for Primary Lysis of Acute Pulmonary Thromboemboli (PE) in Rabbits
  • Petr Klement, Henderson Research Centre, Hamilton, Canada
  • The Antifibrinolytic Effect of Heparin - Exacerbation by Carboxypeptidases
  • John B Walker, Henderson Research Centre / McMaster University, Canada
  • A Study of the Role of Plasminogen, TAFIa, and High Molecular Weight Fibrin Degradation Products on the Rate of tPA Inhibition by PAI-1
  • Mr Mark M Schneider, Queen's University, Canada
  • Decreased Activity of Thrombin Activatable Fibrinolytic Inhibitor in Anticoagulated States: A Possible Explanation for the Observed Profibrinolytic Actions
  • Michelle R Florian-Kujawski, Loyola University Medical Center, United States
  • Migration of the Activation Peptide of Thrombin-activatable Fibrinolysis Inhibitor (TAFI) during SDS-polyacrylamide Gel Electrophoresis
  • Ana HC Guimarães, Gaubius Laboratory, TNO Prevention and Health, Leiden, The Netherlands
  • Development of an Elisa Exclusively Reacting with the Released Activation Peptide from TAFI (proCPU)
  • Erik PJ Ceresa, KULeuven, Belgium